Equine Viral Arteritis Outbreak in Argentina

C. OLGUIN PERGLIONE; M. CÓRDOBA; G. ECHEVERRIA; P. TIMONEY; S. TORDOYA; F. DARQUI; G. METZ; S. MIÑO; L. BECERRA; M. SERENA; A. VISSANI; T. GONZALEZ; M. SILVESTRINI; S. CORVA; L. UNCAL; J. DAYRAUT; A., BADARACCO; M. BARRANDEGUY

Proc Annu Meet Unit States Anim Health Assoc

Genamics Journal SEEK

Año: 2010

0082-8750

In 1984, Nossetto et al. (1984) were the first to report serological evidence of EAV infection in Argentina, finding 23 out of 250 (9.2%) warmblood horses positive for antibodies to the virus. In 1998, several EAV-antibody positive animals were detected on two sport-horse breeding farms that practiced artificial insemination using imported semen. Although the National Animal Health Authorities (SENASA) carried out an epidemiological survey, the original source of the virus could not be identified (Echeverria et al., 2003). A follow-up study on one of those farms between July 2001 and December 2003 revealed a prevalence of 45.8%; the carrier state was detected by virus isolation in three out of four stallions on the premises (Echeverria et al., 2007). In addition, EAV was reportedly isolated from the testicle of a seropositive stallion that had been imported to Argentina in 1998 (Metz et al., 2008). The testicle, which had been collected in 2000, had been stored at -20º C for several years. Prior to this year’s occurrence of EVA in Argentina, the virus had not been previously involved with respiratory disease, abortion or foal death. The prevalence of EAV infection was low and restricted to sport-horse breeds, and in some cases, to certain breed lineages (Vissani et al., 2008).