INVESTIGADORES
LUQUET Carlos Marcelo
artículos
Título:
Acid‑base balance and ionic regulation during emersion in the estuarine intertidal crab Chasmagnathus granulata. Dana (Decapoda, Grapsidae)
Autor/es:
LUQUET, C; ANSALDO, M.
Revista:
COMPARATIVE BIOCHEMISTRY AND PHYSIOLOGY A-MOLECULAR AND INTEGRATIVE PHYSIOLOGY
Editorial:
Elsevier
Referencias:
Lugar: Amsterdam; Año: 1997 vol. 117 p. 407 - 410
ISSN:
1095-6433
Resumen:
Haemolymphatic PO2, PCO2, and pH, and ionic concentrations of haemolymph and branchial chamber water were measured in submerged and emersed crabs. Hypercapnia and respiratory acidosis were recorded after 15 min of air exposure, then PCO2 remained constant and pH was compensated. Sodium concentration increased in haemolymph and decreased in branchial water. The difference between haemolymph Na1 and Cl2 increased dramatically. Acid–base balance is restored by two mechanisms: a) increase of the PCO2 gradient across the gills and consequently the rate of CO2 excretion; b) the increase in the strong ion difference, caused by branchial sodium uptake, compensates the hypercapnic acidosis.O2, PCO2, and pH, and ionic concentrations of haemolymph and branchial chamber water were measured in submerged and emersed crabs. Hypercapnia and respiratory acidosis were recorded after 15 min of air exposure, then PCO2 remained constant and pH was compensated. Sodium concentration increased in haemolymph and decreased in branchial water. The difference between haemolymph Na1 and Cl2 increased dramatically. Acid–base balance is restored by two mechanisms: a) increase of the PCO2 gradient across the gills and consequently the rate of CO2 excretion; b) the increase in the strong ion difference, caused by branchial sodium uptake, compensates the hypercapnic acidosis.CO2 remained constant and pH was compensated. Sodium concentration increased in haemolymph and decreased in branchial water. The difference between haemolymph Na1 and Cl2 increased dramatically. Acid–base balance is restored by two mechanisms: a) increase of the PCO2 gradient across the gills and consequently the rate of CO2 excretion; b) the increase in the strong ion difference, caused by branchial sodium uptake, compensates the hypercapnic acidosis.1 and Cl2 increased dramatically. Acid–base balance is restored by two mechanisms: a) increase of the PCO2 gradient across the gills and consequently the rate of CO2 excretion; b) the increase in the strong ion difference, caused by branchial sodium uptake, compensates the hypercapnic acidosis.2 increased dramatically. Acid–base balance is restored by two mechanisms: a) increase of the PCO2 gradient across the gills and consequently the rate of CO2 excretion; b) the increase in the strong ion difference, caused by branchial sodium uptake, compensates the hypercapnic acidosis.2 excretion; b) the increase in the strong ion difference, caused by branchial sodium uptake, compensates the hypercapnic acidosis.