Sodium bentonite and monensin under chronic aflatoxicosis

A. MAGNOLI; M. TEXEIRA; ROSA CA; R. D. MIAZZO; L. R. CAVAGLIERI; C. E. MAGNOLI; A. M. DALCERO; S. M. CHIACCHIERA

POULTRY SCIENCE

POULTRY SCIENCE ASSOC INC

Año: 2011 p. 1 - 7

0032-5791

  sites on the clay surface and effectively displaces An in vivo experiment carried out with poultry showed productive or biochemical parameters. However, liver 1 (AFB1). In vitro studies demonstrated that the above NaB wasClay feed additives have been increasingly incorporated into animal diets to prevent aflatoxicosis. Due to the nonselective nature of the binding interaction, many important components of the diets could also be made unavailable because of these feed additives. The anticoccidial monensin (MON) could also be sequestered by these clays. The use of sodium bentonite (NaB) from a mine in the province of Mendoza, Argentina, was investigated as a sequestering agent to prevent the effects of 100 ìg/kg of dietary aflatoxin B 1 for the adsorptiona good candidate to prevent aflatoxicosis. They also showed that MON competes with AFB the toxin when it is in low concentration. Even though the levels of MON in diets, approximately 55 mg/kg, are high enough to not be significantly changed as a consequence of the adsorption, they can further affect the ability of the clays to bind low levels of AFB1 t1 does not significantly change at 100 ìg/kg of AFB1 histopathology not only confirmed the ability of this particular NaB to prevent aflatoxicosis but also the decrease of this capacity in the presence of 55 mg/kg of MON. This is the first report stressing this fact and further research should be performed to check if this behavior is a characteristic of the assayed NaB or of this type of clay.